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DMSO and Musculoskeletal Injuries
Many of the early adopters of DMSO went from skeptics to believers because of the rapid and dramatic improvements they saw from it healing acute injuries (e.g., as they had patients with debilitating bursitis in the shoulder recovering within minutes of receiving DMSO).
As it was far safer and more effective than any other way to treat musculoskeletal injuries, joint disorders or chronic pain, DMSO was rapidly adopted by doctors and pharmaceutical companies across the country (e.g., millions were invested to bring DMSO products to market and hundreds of thousands of Americans had life-changing benefits from it).
Unfortunately, as DMSO's use was skyrocketing, on November 10, 1965, the FDA decided to globally ban all research on it by falsely claiming it was incredibly dangerous. Because of this, there was an explosion and then sudden disappearance of DMSO research, which sadly continued even with Congress repeatedly trying to get the FDA to overturn their indefensible prohibition of DMSO.
Note: A detailed review of DMSO's extensive safety data and toxicology studies can be viewed here.
As a result, very little knowledge now exists of DMSO's use in human musculoskeletal injuries other than it existing in a few FDA approved products (where it typically is combined with another agent). Remarkably however, it is fully permitted in veterinary medicine (which led to a lot of Americans using DMSO that was "meant for horses") where it is frequently utilized for musculoskeletal injuries. Those forgotten studies include:
| A 1964 study,31 where 22 out of 25 patients with subacromial bursitis experienced a rapid improvement within 30 minutes of DMSO, while in chronic cases 32 of 40 patients improved and in some patients, a reduction in shoulder calcium deposits was also noted (which in a later 1967 study,32 were shown on X-ray to disappear following DMSO). |
This 1965 study:33
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This large 1967 study:34
![]() Note: In that study,35 many of the results were immediate and dramatic. For example, this was one bursitis patient. ![]() |
This 1967 study:36
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This 1967 study:37
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A 1967 study that found:38
![]() Note: The miscellaneous conditions treated in this study by DMSO included 19 cases of sciatica, 6 cases of coccydynia, and 2 cases of lupus.39 |
Another 1967 study40 found:
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| A 1967 study41 gave PT and 70% DMSO to 7 people with frozen shoulders, 4 of whom had excellent pain relief and improved motion. |
A 1967 blinded study42 for acute musculoskeletal disorders (using 10% DMSO gel as a "placebo") that found:
![]() ![]() In that study, its author (a former president of the Aerospace Medical Association43) remarked:
He then conducted a follow-up double-blind study44 on patients with sprains, strains, bursitis, or tendinitis which found DMSO significantly improved those conditions and reduced the time patients lost from work. |
| A 1994 blinded study45 gave 157 patients with acute tendinopathies (e.g., tennis elbow) 10% DMSO gel or a placebo ointment three times a day for 14 days within 3 days of symptoms starting. Pain of movement under loading and the mobility of the joints were significantly improved after, respectively, 3 and 7 days of treatment with DMSO. After 14 days on DMSO, a further improvement was observed, and 44% of the patients (and 9% of placebo) were pain-free.
Note: DMSO has also been reported to be effective for carpal tunnel syndrome46 (and other hand issues like trigger fingers). For those struggling with carpal tunnel syndrome, I discussed our approaches to the disorder here. |
Finally, a 1967 analysis47 of 76 studies using topically applied (90%) DMSO for musculoskeletal conditions found 72% improved. Specifically:
![]() ![]() ![]() Note: The review also included 102 Traumas (contusion, fracture, etc.), 29 Tenosynovitis, 27 Neuritis, 20 Muscle spasms, 20 unspecified types of arthritis, and 220 miscellaneous issues (e.g., fibrositis, epicondylitis, synovitis, calcific tendinitis). To quote the authors:
Finally, at a symposium on DMSO,48 data on 9,521 patients were presented, which showed DMSO was effective therapy in a wide variety of acute traumatic conditions, in acute and chronic subacromial bursitis, osteoarthritis, gouty arthritis, and in some patients with rheumatoid arthritis (along with other conditions such as early Dupuytren's contracture). Note: A later 1981 study49 also found DMSO was superior to indomethacin in the treatment of gout. |
Sports Injuries
"What I like about DMSO is that you don't have to interrupt your training every time you get a minor pull or sprain. It doesn't pump you up like certain pills. It's simply a very useful thing to use for simple athletic injuries.
Some people have told me that you shouldn't use it because it might mask the pain of a serious injury, but a good athlete knows his body well. Even when I'm using DMSO, I know when I can push and when I can't." — Al Oerter, a discus thrower and the first American to win 4 consecutive Olympic gold medals.50
One of the greatest challenges professional athletes face are sports injuries which prevent them from returning to the field, particularly since many sports injuries are a product of micro-injuries building up until a critical point is passed (e.g., from adhesions and scars in the soft tissue).
In turn, since DMSO both heals micro-injuries and rapidly treats traumatic injury (returning them to full functionality), DMSO was rapidly adopted by professional athletes once they realized what it could do for their careers (and being off the field was often devastating to their careers).
In turn, due to the voice their position afforded them, a few professional athletes (e.g., Atlanta Falcons Quarterback June Jones51 — who now is a coach52) became some of the most impactful advocates for DMSO (e.g., Jones stated in Congressional testimony53 that "veterinary" DMSO was widely used but athletes were afraid of publicly discussing it). Likewise, in 2013, a Dallas Cowboys Lineman stated:54
"You get it [from] the veterinarian and it goes right to the bloodstream. It's an ointment that's like anti-inflammatory. You put it on your skin and you put it on a muscle, and I guarantee you, in about 30 minutes you'd feel it. It wasn't on the list [of banned substances] … we used DMSO and people knew it. Everyone knew about it."
Furthermore, in his riveting testimony, Jones provided cases that left the Congressmen in disbelief, such as a teammate with a bone chip and a torn ligament (which would require months of recovery and hence end their season) taking DMSO immediately after the injury and 7 or 8 days later returning to the field (with the bone chip remaining but no longer causing issues).
Likewise, at that Congressional hearing, the former team physician for the Oakland Raiders55testified that he'd used 70% topical DMSO on a careful and controlled basis for his players 20 to 30 times a year for 5 years. From this, he observed that DMSO was the most beneficial when given in the first 3 to 4 days of an acute injury where a muscle or joint had severe swelling, particularly of the extremities, especially the ankle, elbow, hands, or wrist.
Overall, he stated that DMSO provided good to excellent results 70% to 80% of the time (e.g., through reduced pain and swelling) and the players felt they were able to return to play 50% to 75% faster than they had from similar injuries in the past. Conversely, they did not find DMSO was helpful for chronic injuries, but this may have been due to it not being used long enough for the effects to kick in.
Note: He also emphasized that DMSO would transform the field of occupational medicine. I fully agree with his assessment, especially given just how frequently Worker's Comp fails to help its patients.
Similarly, podiatrist Lowell Scott Weil (who was the physician for both the Chicago Bears and the United States Olympic gymnastics team) used DMSO on a regular basis (particularly injured gymnasts).
After 12 years of using it, he shared,56 he'd seen it rapidly heal injuries (e.g., he had a gymnast who suffered an ankle sprain expected to end her season, but instead quickly recovered and made the U.S. Olympic team, and a football player who tore his hamstring but was able to rapidly return to the field).
Overall, he had a 60% treatment success rate and saw the best response to DMSO for tendinitis, myositis, and post-injury situations such as muscle pulls, ankle sprains, strains, and tears of the soft tissue (and conversely the only side effects he had were skin irritation). Additionally, he also used it for arthritic patients (especially rheumatoid arthritis) with many having dramatic relief. Many other compelling anecdotes exist.
For example, this book57 discusses the experience of an Oregon State track coach and early adopter of DMSO who had many amazing stories of DMSO treating hamstring and achilles tendon injuries such as an athlete being able to return to the field at full capacity 3 days after a normally disqualifying hamstring injury and the story of a blind long distance runner who was able to run due to DMSO fixing musculoskeletal injuries and (according to the author) then played a pivotal role in opening the sport to women.
Note: A major problem in certain sports like football is repeated concussions (which are now recognized to put them at risk for cognitive impairment and dementia later in life). As discussed in the first part of this series, in addition to treating strokes and spinal cord injuries, DMSO is also immensely helpful for mitigating the effects of concussions.
Research also directly demonstrates DMSO's utility in sports medicine:
• A 1965 study58 treated 47 injured athletes from a wide range of sports (e.g., tennis, diving, or wrestling) by applying 90% DMSO applied to the injured areas 3 times a day initially and then after 2 days, twice a day. The 30 acute traumas (e.g., sprains, strains, dislocations, serious cuts) were observed to rapidly resolve, sometimes "so spectacularly as to compel us to urge our patients to observe greatest caution in order to avoid further damage to a joint."
The 10 chronic conditions (e.g., tennis elbow) and 7 conditions resulting from prolonged immobilization also responded rapidly and those athletes were often able to quickly return to the field. These results and the lack of observed adverse events led the investigators to argue DMSO urgently needed to become the standard of care in sports medicine.
• A study59 of 78 patients (mostly athletes) with overstrained tendons received Dolobene gel (15% DMSO, dexpanthenol and heparin) for 2 to 3 weeks, with over 50% having a significant improvement of symptoms and those improvements including a 94% improvement in pain, a 55% improvement of swelling, 95% improvement of redness and 92% improvement of warmth.
• A study60 gave Dolobene gel to 30 athletes with soft tissue injuries of the upper and lower extremities twice daily for 4 weeks. There were 4 athletes with contusion of the shoulder, 8 with distortion and contusion of the knee joint, 8 with muscle, tendon and ligament lesions, and 10 with distortion of the ankle joint.
Following DMSO, 10 had an excellent response (improvement), 5 had an excellent to good response, 10 had a good response and 5 had a moderate response. Specifically, pain, inflammation, swelling, reabsorption of hematomas, tenderness and recovery time were assessed.
• A study61 gave Dolobene gel and ultrasound to 15 subjects who had received a blunt tissue trauma (without fracture) to the lower extremity within the last 24 hours. Compared to 15 placebos, the treatment resulted in a faster relief of pain, reduction of edema, and recovery of mobility.
• A 1966 study62 of 28 professional baseball players found that giving them DMSO after injuries caused their downtime be one third of what was observed by the treating physician in the previous year with 42 players.
Note: While not quite the same as getting tackled, I've also come across cases63 of individuals taking DMSO immediately after getting hit by a car while crossing the street (which caused injuries but no fractures) and immediately fully recovering.
Conclusion
Many of the benefits of DMSO are so extraordinary that they understandably invite a healthy degree of skepticism, and it is for that reason I have spent months carefully compiling the evidence behind it. Likewise, after presenting the initial case for DMSO to the readership of the Forgotten Side of Medicine, I put out a call for what those who'd tried DMSO had experienced.
In those comments, dozens of readers reported remarkable experiences of DMSO, many of which mirror those described throughout this article, but also other even more remarkable ones (e.g., for a child with Down Syndrome, a man with Parkinson's and a woman who had severed her spinal cord).
The suppression of DMSO has always deeply bothered me, and in turn, I feel incredibly grateful to be alive at a time when the world is ready to learn of the suppressed medical truths many before me (e.g., the DMSO researchers) devoted their lives to bring to humanity.
Furthermore, I believe this is just the start, as beyond open platforms like Twitter (X) rapidly eroding the public's trust in corrupt medical dogmas, I know through trusted confidants directly connected to RFK Jr. that the next four years offers an unprecedented opportunity to begin rectifying many of the previously insurmountable problems that have plagued our health care system and make America Healthy Again.
Author's note: This is an abridged version of a longer article that goes into greater detail on the points mentioned here, others not as extensively covered (e.g., the wealth of evidence DMSO is a life-changing pain treatment), and guidance for topical DMSO use (e.g., dosing, therapeutic precautions and where to obtain it).
That article and its additional references can be read here (along with a companion article discussing DMSO's remarkable utility for a variety of musculoskeletal injuries and chronic pain conditions and an article about how DMSO treats a variety of "incurable" autoimmune and genetic disorders).

























Washington D.C. – October 28, 2025





Treason isn't always a single, dramatic act. Sometimes, it's a slow-acting poison, administered over years to weaken a nation from within.
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White Hats have started housing Deep State detainees closer to home; rather than ship or fly them to overcrowded detention centers like GITMO and Camp Blaz, the Hats are availing themselves of a long-ignored resource: FEMA Camps.




Mebendazole/ Fenbendazole/Albendazole Anticancer pathways and mechanisms A compound originally developed as a treatment for parasitic worms, mebendazole (MBZ) works by fatally disrupting the cellular microtubule formation in abnormal cancer cells that occurs as the cell is attempting to divide. Like the other benzimidazoles, Mebendazole binds to the tubulin colchicine-binding domain and appears to act by both p53-dependent and independent mechanisms. (728) MBZ inhibits many factors involved in tumor progressions such as tubulin polymerization, angiogenesis, pro-survival pathways, matrix metalloproteinases, and multi-drug resistance protein transporters. (729) MBZ inhibits cancer stem cells; this mechanism of action is critical in preventing metastasis. (223, 729) In addition, in a juvenile glioblastoma mouse model MBZ reduced tumor cell growth and invasion when evaluated under in-vitro and in-vivo conditions through inhibition of both the glutaminolysis and the glycolysis pathways. (304) In this study the effect of ketosis and MBZ were synergistic in inhibiting tumor growth. MBZ decreases the activity of the Hedgehog pathway, which is common in gliomas, melanomas, lung cancers, ovarian cancers, and colorectal cancer. (155) MBZ inactivates Bcl-2 and activates caspases to promote apoptosis in cancer cells and the release of cytochrome c which has also been shown to trigger apoptosis in malignant cells. Benzimidazole modulates the typically overactivated MAPK pathway, switching it to activate the apoptotic pathway, rather than the anti-apoptotic pathway; it also destabilizes microtubules, structural proteins required to maintain a cell's integrity during the process of mitosis, among other functions; it also interferes with cancer cells' glycolysis-dependent metabolism, upon which most cancers are heavily preferentially dependent, as well as functioning as an inhibitor of mitochondrial oxidative phosphorylation, or OXPHOS, which reduces the residual energy available via the ordinary metabolic ATP production pathway. MBZ can cross the blood-brain barrier and has been demonstrated to slow the growth of gliomas by targeting signaling pathways involved in cell proliferation, apoptosis, invasion, and migration, as well as by making glioma cells more susceptible to conventional chemotherapy and radiotherapy. (730) MBZ can also sensitize cancer cells to conventional therapy, such as chemotherapeutics and radiation, enhancing their combined antitumor potential, confirming that MBZ may be useful as an adjuvant therapeutic combined with traditional chemotherapy. (730) When combined with low-dose chemotherapy there is also evidence these drugs help to destroy the tumor-associated macrophage cells that may help maintain a favorable environment for the cancer to flourish. Clinical studies The use of benzimidazoles in cancer is limited to a few case reports (731, 732) and a small case series. (733) Mebendazole is a component of the multidrug cocktail used in the METRICS study.(257) The use of benzimidazoles, and in particular fenbendazole, has achieved much attention as a repurposed drug for cancer due to the reported experience of Joe Tippens. (146) In 2016, Tippens was diagnosed with non-small-cell lung cancer with extensive metastatic disease. At the advice of a veterinarian friend, he took Fenbendazole together with nanocurcumin, and three months after starting these drugs his PET scan was completely clear. He remains alive and disease-free up until the present; however, some questions surround his apparent cure. Types of cancers that mebendazole may be beneficial for A wide variety of cancers, including non-small cell lung cancer, adrenocortical, colorectal, chemo-resistant melanoma, glioblastoma multiforme, colon, leukemia, osteosarcoma/soft tissue sarcoma, acute myeloid sarcoma, breast (ER+ invasive ductal), kidney, and ovarian carcinoma, have been shown to be responsive to benzimidazoles, including MBZ. (252, 728-730, 734-743) Dosing and cautions We suggest Mebendazole 100-200 mg/day. The cost of mebendazole in the U.S. skyrocketed once this drug was discovered to have activity against cancer ($555 for a single 100 mg tablet?). However, mebendazole is available from international (India) and local compounding pharmacies for between 25c to $2 for a 100 mg tablet. Ivermectin can be considered an alternative if mebendazole is unavailable. However, it I likely that both drugs combined may have additive or synergistic anticancer activity. 8. Green Tea Anticancer pathways and mechanisms Green tea is a significant source of a type of flavonoid called catechin, which includes epigallocatechin gallate (EGCG), epigallocatechin (EGC), epicatechin gallate (ECG), and epicatechin (EC). The most abundant individual catechin in fresh tea leaves is EGCG, which is more than 40% of the total content of catechins. (234) Green tea catechins (GTCs) have been proven to be effective in inhibiting cancer growth in several experimental models. (744-746) In addition, GTCs may have synergistic anticancer activity when combined with other phytochemicals, particularly resveratrol. (747, 748) GTCs, particularly EGCG, may have a role in both the prevention and treatment of cancers, (749) specifically those dependent on the glutamate pathway as a source of energy. Mitochondrial glutamate dehydrogenase (GDH) catalyzes the oxidative deamination of L-glutamate. Activation of GDH is tightly correlated with increased glutaminolysis. Furthermore, glutamate serves as a mitochondrial intracellular messenger when glucose is being oxidized and the GDH participates in this process by synthesizing glutamate. (750) Li and colleagues demonstrated in vitro that EGCG allosterically inhibits GDH in nanomolar concentrations. (301, 302) GTCs have an important anticarcinogenic role by promoting and/or inhibiting signal transmission through the targeted regulation of multiple links in the signal pathways that are activated or inhibited in cancer cells. (234) EGCG regulates signaling pathways by interacting with membrane receptors. EGCG significantly inhibited the expression of VEGF and reduced VEGF receptors. Inactivation of the VEGF signaling pathway suppresses angiogenesis, a common strategy for inhibiting carcinogenesis. EGCG activates PKA, which dephosphorylates related proteins such as the tumor suppressor Merlin and inhibits the proliferation of cancer cells. (751) EGCG inhibits STAT3 phosphorylation by blocking JAK2 phosphorylation. STAT3 suppresses anti-tumor immune responses and promotes the proliferation and migration of cancer cells. EGCG inhibits the MAPK signaling by competing for the phosphorylation sites of downstream proteins. EGCG inhibits the Wnt pathway by phosphorylating β-catenin and promoting its degradation. EGCG inhibits transcription factors involved in activating the Sonic hedgehog pathway. EGCG inhibited the activities of MMP2 and MMP9 and promoted the expression of tissue inhibitor of MMPs (TIMp1/2) to suppress the invasion and metastasis of tumor cells. (751) Green tea extract has been demonstrated to suppress cancer stem cells. (752, 753) GTCs have anticancer effects via additional pathways. (234) GTCs exert potent and selective in vitro and in vivo pro-apoptotic activity in cancer cells via several pathways. (744, 745, 754) GTC inhibits A549 cells by regulating its cell cycle arrest, increasing the expressions of p21 and p27, and inhibiting the expressions of p-AKT and cyclin E1 in a dose-dependent manner in the cancer cells. (755) EGCG inhibited the proliferation of human lung cancer cells by targeting the epidermal growth factor receptor (EGFR) signaling pathway. GTCs have been demonstrated to alter the tumor microenvironment (TME) thereby attenuating immunosuppression and the risk of metastases. (748) Flavonoids including GTCs (and resveratrol) are potent modulators of pro-inflammatory gene expression being, therefore, of great interest as agents selectively suppressing molecular targets within pro-inflammatory TME. GTCs have been demonstrated to increase the ratio of active cytotoxic T lymphocytes to Tregs in tumors, indicating a switch of "cold" tumors to "hot" with significantly improved anti-tumor immune therapeutics. (756) GTCs have anticancer effects by enhancing anticancer immunity via PD-1 axis and TLR4 pathways. (757, 758) In addition, GTCs repolarize tumor-associated macrophages (M2 to M1 macrophages), triggering an immune response and limiting metastases. (759) GTCs have been demonstrated to attenuate MDSC-mediated immunosuppression and increased the proportions of CD4+ and CD8+ T cells. (760) Studies have shown that 20% of cancer-related deaths were directly due to TLR-induced cancer cachexia, in which cancer cells released heat shock proteins that acted as TLR-4 agonists in macrophages, skeletal muscle, and fat cells, causing downstream signal transduction. EGCG effectively downregulates the TLR-4 signal pathway. (758) GTCs inhibit the accumulation of MDSCs, leading to restoration of the IFN-γ, enhancing the activity of CD8+ T-cells, and improvement of the ratio of CD4(+) to CD8(+) T-cells, which is beneficial to the improvement of the immune system's attack on tumor cells. (234) In addition, a phytochemical mixture including GTCs exerted anti-tumor activity by repolarization of M2-polarized macrophages and induced the production of IL-12, which recruit cytotoxic T lymphocytes and natural killer cells (NK) with the tumor microenvironment. (759) In addition to all these beneficial effects, GTCs potentiate the effects of conventional chemotherapeutic agents. Due to their effects on the important signaling pathways in vivo, catechins are often used as sensitizing agents in combination with chemotherapeutic drugs. The combination of anticancer drugs with catechins, whether before or after drug administration, reduced the toxicity of these drugs and enhanced the clinical efficacy by accelerating apoptosis of cancer cells. (234) Importantly, the combination of a number of chemotherapeutic drugs with GTCs will improve the chemotherapeutic sensitivity of cells to the drug, allowing a reduction in the dose of the chemotherapeutic drug. (234) Clinical studies Numerous experimental models have explored the mechanistic anticancer effects of GTCs; this data is supported by epidemiologic data, a case series of patients with B cell malignancies,(761) several case reports,(762, 763) and a RCT. A meta-analysis including 18 prospective cohorts and 25 case-control studies showed a significant inverse association between intake of tea catechins and risk of various cancers, with a relative risk (RR) being 0.935 (95% CI = 0.8910.981). (234) Similarly an umbrella review and meta-analysis by Kim et al, which included 64 observational studies (case-control or cohort) demonstrated that GTC significantly reduced the risk of gastrointestinal cancer (oral, gastric, colorectal, biliary tract, and liver), breast cancer, and gynecological cancer (endometrial and ovarian cancer) as well as leukemia, lung cancer, and thyroid cancer. (243) In a phase I dose finding study in patients with Chronic Lymphocytic Leukemia EGCG was well tolerated and a decline in the absolute lymphocyte count and/or lymphadenopathy was observed in the majority of patients. (764) Lemanne et al reported on a patient who demonstrated a complete and durable remission of chronic lymphocytic leukemia (CLL) following high dose EGCG. (763) In a randomized, double-blind, placebo-controlled study, treatment with 600 mg/day of green tea catechins reduced the risk of prostate cancer from 30% to 3% in men with high-grade prostate intraepithelial neoplasia. (303) Types of cancers that green tea may be beneficial for Green tea catechins may be effective against a range of tumors including cancers of the prostate, breast, uterus, ovary, colorectal, glioma, liver and gallbladder, melanoma, and lung cancers. (234) GTCs appear to be particularly beneficial for prostate cancer as well as breast cancer. (299, 303, 744-747, 760, 765) Dosing and cautions Green tea catechins should be taken in a dose of 500-1000 mg/day. Green tea extract should be taken during/after a meal rather than on an empty stomach. (244) Green tea extract has rarely been associated with liver toxicity. (766) The safety of green tea extract was evaluated by the US Pharmacopeia (USP) Dietary Supplement Information Expert Committee (DSIEC). (244) The DSIEC concluded that "when dietary supplement products containing green tea extracts are used and formulated appropriately the Committee is unaware of significant safety issues that would prohibit monograph development." (244) Based on this data we suggest that green tea exacts be taken in the dosages recommended by the manufacturer. Regular liver function tests are suggested in patients taking green tea extract and green tea exact should be avoided/used cautiously in those with underlying liver disease.
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God said "His David" DJT would win, and God keeps His Word. GOD saved him from 30+ assassins. Thank You Father, Son, Holy Spirit. God spoke through His prophets, Kim Clement, Julie Green, Rick Joyner, Dutch Sheets, Joseph Z, Lance Wallnau, Brandon Biggs, Amanda Grace. AMERICA PRAYED. GOD ANSWERED. Also, God said He would expose and remove every evil person in government, media, judicial, medical, pharma, Hollywood, WHO, United Nations, the Fed, voting structures and corporations. HE WILL PERFORM HIS WORD. HE IS DOING THIS NOW.
Voter fraud in 2020?
















Composite of Polls



























THE PASSION OF CHRIST:
Pastor Tim Keller Daily Prayers – 
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